Home Dermatitis: Stasis Dermatitis (SD)

Dermatitis: Stasis Dermatitis (SD)


Defining Characteristics

Xerosis, erythema, red-brown discoloration from hemosiderin deposits and degraded extravasated RBCs, dilated superficial veins; often involves medial ankle, possible lipodermatosclerosis (hard feeling from underlying fat necrosis); hypopigmentation, ulceration

Disease Development

Abnormal circulation in skin comprises the skin barrier, causing dryness and inflammation –> itch –> LSC & SD
OLD theory – SD caused by stasis and hypoxia, but pts actually have high flow rate and oxygen
Abnormal microcirculation – increased permeability of dermal capillaries allows leakage of fibrinogen, which polymerizes to fibrin to form fibrin cuff around capillaries –> ultimately

Risk factors

Venous insufficiency

Conventional Treatment

Often require hospitalization for tx of venous ulcers


Can be complicated by LSC